Judson, G.J
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Laboratory research demonstrates that BPC-157 treatment accelerates wound healing in diverse injury models including alkali burns, surgical incisions, and chronic wounds, with the peptide enhancing proliferation, migration, and angiogenesis in vitro through dose-dependent ERK1/2 phosphorylation leading to increased cellular proliferation, migration, and vascular tube formation
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Investigators observe how this pentadecapeptide interacts with focal adhesion kinase (FAK) and paxillin cascades to physically guide structural cells to wound sites, balancing local nitric oxide signaling to stabilize endothelial cell survival curves
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