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5I, S2B), indicating that inducing ferroptosis by targeting x c can increase the sensitivity of ER+ breast cancer cells in response to endocrine therapy

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Endotoxin testing helps identify the presence of bacterial byproducts that could introduce unwanted variables into laboratory investigations

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Deletion of p53 promotes the interaction of DPP4 with nicotinamide adenine dinucleotide phosphate oxidase 1 (NOX1), which forms the NOX1-DPP4 complex and mediates plasma membrane lipid peroxidation reactions and ferroptosis