doi: 10.3389/fimmu.2020.01451 Received 09 April 2020 Accepted 04 June 2020 Published 19 June 2020 Volume 11 - 2020 Edited by Xulin Chen, Jinan University, China Reviewed by Vincent T
In this context, it has been demonstrated that the restoration of -cell mass can occur not only through proliferation of existing -cells but also via transdifferentiation of other pancreatic cell types, such as -, -, PP-, and ductal epithelial cells (Figure 1B)
It's the dividing line between valid data and wasted resources
PMC 7266692
Well-established contributors include microbial translocation due to gut barrier impairment, residual viral replication in sanctuary sites, reactivation of latent co-infections (e.g., cytomegalovirus), and increased cellular senescence ( This review aims to synthesize current knowledge on how HIV/SIV infection and ART induce mtDNA damage and how this damage acts as a key indicator and mechanistic driver of associated immunometabolic changes
First read: Can I combine peptides in the same syringe