As a peptide derived from human gastric juice, it is natively resistant to low pH and proteolytic enzymes
Key cited studies: Cole LA (2012) hCG, Five Independent Molecules Clin Chim Acta 413(12):4865
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In mouse models of highfat dietinduced liver steatosis, it significantly inhibits hepatic NNMT activity, reduces NAM methylation, increases NAD+ and SAM levels, enhances mitochondrial fatty acid oxidation, reduces hepatic triglyceride and lipid accumulation, lowers lipotoxicity markers (e.g., malondialdehyde, transaminases), improves hepatocellular injury, and reverses steatosis