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In vivo, systemic NGB, myoglobin (MB), and CYGB administration in CCl-induced fibrosis dampened inflammation and slowed fibrogenesis without toxicity in treated mice, indicating a feasible safety window and a globin-based therapeutic class concept for fibrotic liver disease [112] (Figure 5)

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Discussion This review covers the importance of maintaining NAD + levels for cellular homeostasis and mitochondrial maintenance, as well as evaluating established and novel NAD + boosting strategies from simple supplementation of precursors to modulation of enzymes by drugs

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