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Numerous treatment approaches have focused on modifying the system Xc-/GSH/GPX4 axis because of its significance in controlling ferroptosis

We next optimized a NRF2 transcriptional signature resulting in a set of 59 genes based on their contribution to the model and overlap with previously reported NRF2 signatures (Figs

A lipoprotein produced in the liver and the brain

Together, these findings position GSH not only as a guardian of cellular integrity but also as a context-dependent determinant of cell death susceptibility and therapeutic vulnerability

*Correspondence: Min Liu, [email protected]

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