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Thyroid, 2020;30(10), 1440-1450.7 Kubiak M, Wiatr K, Kowalczyk A, Grecki R, Gajewska D, Sobieska M

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Mechanisms such as cellular senescence, genomic instability, telomere attrition, mitochondrial dysfunction, epigenetic alterations, and loss of proteostasis all contribute to an aging phenotype (Tenchov et al 2024) and are considered hallmarks or biomarkers for aging

found that epithalon elongated telomeres and induced telomerase activity when introduced to telomerase-negative human fetal fibroblast cultures, with the authors concluding that epithalon may prolong the lifespan of cell populations [2]

The participants had an average age of 49