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They wrote that the peptide seemed to stimulate, either directly or indirectly, a specific gene involved in the bodys response to injury
Kabir AMR, Inoue D, Kakugo A
135 ZengH.SpencerN
doi: 10.1111/j.1368-5031.2006.00825.x
The explanation for this discrepancy can be found in recent papers showing that some cancer cell linesincluding HCT116are not affected by L-BSO or GSH synthesis inactivation because they can compensate GSH depletion by the thioredoxin (TXN) pathway 25 , and by maintaining protein homeostasis through deubiquitinating enzymes (DUB) 32
[3] Other populations may experience effects at lower or higher dosages depending on differences in P-450 enzyme activity and other factors which affect the amount of NAPQI produced
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