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(8) Under LT stress circumstances, exogenous GSH most prominently G2C concentration may have a potential role in increasing GSH redox and suppressing ABA-mediated signaling in Pusa sheetal cv

Erastin or sorafenib combined with small doses of cisplatin can effectively inhibit the growth of cisplatin-resistant NSCLC cells by inhibiting the Nrf2/SLC7A11 pathway (Li et al., 2020)

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Kraemer et al., 2020) and altering the expression of thousands of genes, many of which are involved in the regulation of skeletal muscle structure, fiber type (Dubois et al., 2013), and mRNA transcription (MacLean et al., 2008) (Figure 1)

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