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However, C16-carnitine disrupts this interaction, resulting in inefficient uptake of Ca 2+ released from the SR by mitochondria, thereby exacerbating intracellular calcium overload [31]

Nrf2-dependent antioxidant pathway Allicin was also found to upregulate the expression of glutamate-cysteine ligase (GCL), the rate-limiting enzyme in glutathione synthesis, and other Phase II detoxifying/antioxidant enzymes, likely via the activation of the nuclear factor E2-related factor 2 (Nrf2)-dependent pathway (19)

A full medical assessment is carried out to identify any contraindications or medication interactions.

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