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Sinensetin attenuates hepatic ischemia-reperfusion injury through suppressing grp78/chop-mediated endoplasmic reticulum (Er) stress in mice

The mechanism does not involve receptor downregulation or tolerance in any known way, but the signal-to-noise ratio is simply too low based on available evidence

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It also activates the Akt survival pathway, reducing apoptosis in stressed cells throughout the body

By contrast, rats bearing GH-secreting tumors displayed significant glomerular hypertrophy and interstitial edema but no tubular hypertrophy, resulting in a 1.6-fold increase in kidney weight in relation to body weight compared to controls [95]