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The 48-h cARLA treatment resulted in mature brain endothelial cell morphology with continuous and strong claudin-5 junctional protein immunostaining at the cell borders (Fig
DA activates the DA receptors on glial cells upon binding, leading to ROS generation and release of pro-inflammatory cytokines from glial cells, triggering neuronal damage [138,139,140]
This perspective takes a look back at the contributions of this and related work in the context of plant biology research at the time, and considers the importance of the pathway within our current understanding of reactive oxygen species biology and redox homeostasis and signalling
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[Abstract] H460 and H1299 cells were treated with erianin with or without the ROS scavenger GSH (1 mM) for 24 h, and cell viability was assayed
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