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In neuronal senescence model studies, the active ingredient of the product can cross the blood-brain barrier (and act directly on neurons in vitro), increase NAD+ levels in nerve cells, protect mitochondrial function, reduce neuronal apoptosis, and promote the synthesis and release of neurotransmitters (such as dopamine and acetylcholine), thereby improving signal transduction in nerve cells

Are there side effects with GLP-1 therapy, and how can genetic testing help

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FASN-dependent lipid metabolism links neurogenic stem/progenitor cell activity to learning and memory deficits

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