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Effects of iron overload on mitochondrial functions Although mitochondria are the major consumers of iron, excess iron in the mitochondrial matrix can result in the generation of ROS and subsequent oxidative damage to the mitochondrial membrane

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Several peptides have been shown to support and stimulate the bone healing response and have been proposed as therapeutic vehicles for clinical use

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A high intracellular ratio of reduced glutathione to oxidized glutathione (GSH/GSSG ratio) is protective of microtubules in their polymerized state by the preventing oxidation of cysteine residues in tubulin subunits