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Whereas the dose plays a role, the 7-day half-life of T 4 allows physiologic compensation, such as the suppression of T 4 to T 3 conversion, inhibition of endogenous hormone production by negative feedback loops, and downregulation of thyroid hormone receptors

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113 Indeed, investigation of glucose kinetics and insulin secretion after a low-CHO meal (20E% CHO, 65E% fat) in five young adult baboons revealed minimal perturbations of glucose homeostasis, in stark contrast to a high-CHO (65E% CHO, 20E% fat) meal