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Its zinc-binding region directly interacts with Cul5, specifically targeting the host antiviral restriction factor APOBEC3G (A3G) for ubiquitination and degradation

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1d was driven by bioisosteric replacement of the fragments bound to subpockets A-C, scaffold-hopping, de novo design, and medicinal chemistry and was facilitated by SZMAP/GamePlan 37 , vBrood 38 , and MOE software 39