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Mechanistically, it is described as a monoamine reuptake inhibitor, affecting: Dopamine Norepinephrine Serotonin In weight-management trials, its primary investigational role involved: Appetite regulation Reduced caloric intake Altered reward-driven feeding behavior It is not a GLP-1 analog and does not function via incretin pathways

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There was a significant average paired fold change in the levels of several GPs 1 week after 4-week intermittent fasting compared with the levels at the end of 4th week during 4-week intermittent fasting (Supplementary Table S3)

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Obesity, hypertension, and chronic kidney disease