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doi: 10.1016/j.devcel.2019.10.007 54 AbramsRPCarrollWLWoerpelKA

These findings imply a significant positive effect with age and accelerate inflammation: following the step of initial dysbiosis and inflammation cascade, the altered GM and persistence of inflammation may act synergistically, prolonging the proinflammatory status

Finally, partial inhibition of GSH may induce stress responses in cancer cells, leading to the upregulation of detoxifying enzymes and drug resistance

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