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H 2 O 2 induces hepatocyte premature senescence via the p53/progerin-dependent pathway To further delineate the molecular mechanism of oxidative stress-induced cell premature senescence, primary hepatocytes, isolated from normal rats, were cultured and stimulated with H 2 O 2 at the different doses (0, 10, 50, 100, 200 nM) for 24 h or at the dose (200 nM) from 0 to 24 h

[62] Ingestion of alcohol can initiate systemic pro-inflammatory changes through two intestinal routes: (1) altering intestinal microbiota composition (dysbiosis), which increases lipopolysaccharide (LPS) release, and (2) degrading intestinal mucosal barrier integrity thus allowing LPS to enter the circulatory system

doi: 10.1021/jf0525941 583 LazarusSAHammerstoneJFAdamsonGESchmitzHH

In the late 1980s, bioactive GLP-1 peptides were purified from gut extracts and were identified to be GLP-1(737) and GLP-1(736)-amide

COX-2 is normally inactive, but can be turned on during inflammation to produce pro-inflammatory prostaglandins