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The anti-inflammatory effects of GLP-1 receptor agonists appear to operate through pathways that are activated at lower receptor occupancy levels than those required for metabolic effects

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Glutathione levels naturally decline with age, stress, poor diet, alcohol consumption, and environmental toxin exposure

In mice lacking SGLT1, peak glucose-triggered GLP-1 and GIP levels were impaired (Gorboulev et al., 2012), and L cells showed no calcium response to glucose intake (Parker et al., 2012a)

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