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Chronic METH exposure reduced acetylated histone H4 levels at the GluA1 , GluA2 , and GluN1 promoters, while increasing HDAC2 and SIN3A binding at GluA1 and GluA2 , suggesting suppressed glutamate receptor expression through epigenetic mechanisms 80
Thoene J, Lemons R, Anikster Y, Mullet J, Paelicke K, Lucero C, Gahl W, Schneider J, Shu SG, Campbell HT (1999) Mutations of CTNS causing intermediate cystinosis
Koeberle, S
Histopathological parameters assessed included neutrophil numbers and neovascularization
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1996 Jul;48(5):439-46
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