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Prevented weight loss in mice during the 24-day treatment period

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It enhances neurotrophic factor expression, supports neural progenitor cell survival, and modulates microglial phenotypes for an anti-inflammatory state, promoting neural repair and regeneration [81]

In mice fed a high choline diet, myocardial hypertrophy induced by TMAO was achieved through the involvement of transforming growth factor-1 (TGF-1) and small mothers against decapentaplegic (Smad), an intermediate effector molecule in the TGF- superfamily signaling pathway (95)

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430 This therapy generates and expands a patients autoimmune T cells in vitro, allowing them to more efficiently recognize specific antigens expressed by tumor cells