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N., Booher, S., Gilron, I., Parada, S

The authors described these structural models as the basis for rational development of p53 inhibitors targeting the senescent cell biology field and diseases linked to cellular senescence including cancer

Studies have included patients ranging from young adults to those over 75 years, with no clinically relevant age-related differences in pharmacokinetics

Critical role of cathepsin L/V in regulating endothelial cell senescence

GDH activity is also increased in tumor cells

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