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Under aerobic condition, SOD overexpression boosted the growth of S-3/pKLH167, S-3/pKLH337, S-3/pKLH341, and S-3/pKLH344 with 1 mmol/L H 2 O 2 treatment compared with the control strain S-3/pKLH32 (Figure 4A)

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Reported safety signals are mild in rodent studies, with no liver, heart, or dependence problems observed

The aforementioned activation may consequently result in the release of substance P, neurokinin A, or calcitonin gene-related peptide (CGRP) [16]

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