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Moreover, phospho-BCL-XL (Ser62) stabilizes G2 arrest by binding to and co-localizing with Cdk1 (cdc2) in nucleolar structures to avoid rapid nucleolar disassembly associated with mitosis onset, playing an unexpected role in the G2 phase checkpoint downstream of DNA damage distinct from its anti-apoptotic function 22

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Upon MT1 binding, the receptor is thought to activate adenylyl cyclase, converting ATP to cyclic adenosine monophosphate (cAMP)

Beyond the CNS mechanisms that reduce food intake, Samms et al