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In mice exposed to cigarette smoke over 12 weeks to induce pulmonary emphysema, GHK-Cu reduced markers of both inflammation and oxidative stress, again via the same NF-B-suppressing, Nrf2-boosting mechanism, this time in a chronic disease model rather than an acute one

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Ultimately, this paper strongly suggests that biological youth or cognitive rescue in the brain is not a singular molecular destination

doi: 10.1155/2022/1582432

[20] [21] In the dermal wounds of rabbits GHK-Cu facilitated wound healing, causing better wound contraction, faster development of granular tissue and improved angiogenesis