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More specifically, the researchers posited that the peptide may have enhanced the expression of Ki-67 and CD98hc that are less intensively synthesized during cell aging. Epithalon also seemed to inhibit the activity of Caspase-3, a crucial enzyme believed to facilitate apoptosis (programmed cell death)

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However, dendritic spine loss in the CA1 region of the hippocampus appeared as early as 4.5 months, with memory deficits becoming evident at 9 months (Hsiao et al., 1996

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