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Thierry AR

Other risk factors that may increase vulnerability include: Long duration of diabetes (typically more than 10 years) Poor glycaemic control prior to starting GLP-1 therapy (HbA1c significantly above target) Rapid reduction in HbA1c (a drop of more than 2 percentage points [22 mmol/mol] within 36 months) Concurrent hypertension , especially if poorly controlled Diabetic kidney disease (nephropathy), which often correlates with retinopathy severity Previous history of retinal treatment (laser photocoagulation or intravitreal injections) People with type 2 diabetes but no retinopathy appear to have minimal risk of retinopathy progression based on current evidence

Semaglutide and Tirzepatide work by increasing insulin production and lowering glucagon secretion as well as targeting areas in the brain that regulate appetite and food intake

Genetic Predispositions in Peptide Pathways and Treatment Response Variability Individual responses to semaglutide vary significantly

Patients can typically return to their daily activities immediately after treatment

Additionally, GLP-1 agonists may modulate the renin-angiotensin-aldosterone system (RAAS), further influencing vascular tone and blood pressure regulation (54)