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In the present study this was evident through distinct growth inhibition, browning and over all degeneration as visualized Haematoxylin staining

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the differences in timing and nature of the stressors, interventions and outcome measures), most of the interventions were able to modulate the ES-induced behaviors

This is the critical finding

Chronic overactivation of HIF1, increased cytoplasmic and mitochondrial calcium overload, and other dysregulations of mitophagy exacerbate ischemia-reperfusion injury