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5a), approved for the treatment of amyotrophic lateral sclerosis, increases ROS levels by multiple mechanisms, including decreasing LDH-A and NAD + levels to increase oxidative stress, as well as interfering with the xCT antiporter to block the cystineglutamate pump

Furthermore, BPC 157 eliminated systemic corticosteroid treatment functionally, macroscopically, and histologically at all investigated intervals [46]

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