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Thalidomide pretreatment markedly upregulated transplanted cell numbers in liver versus non-treatment groups (Viswanathan et al., 2016), also improving engraftment via immune regulation discussed later

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In mouse models, the MDM2-p53-LMNB1 axis also prevented mitochondrial damage and ferroptosis in metanephric tubular epithelial cells in AKI [188]

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63 The accumulated NAPQI depletes GSH in the endoplasmic reticulum to induce an oxidationreduction imbalance, which initiates the phosphorylation of eukaryotic initiation factor 2 (eIF2) to activate ATF6 and CHOP, eventually resulting in hepatocyte apoptosis (Figure 2)

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