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Concurrently, drug development efforts will benefit from atomic-resolution insights into competitive antagonism, allosteric modulation, and ion channel block

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Taken together, our parsimonious model captured the key features that were experimentally observed: calciumdependent and calciumindependent inhibition, exact adaptation, habituation, and the corresponding dynamics in tax6 mutants, which lack inhibitory capacity

GLP-1 receptor signalling promotes -cell glucose metabolism via MTOR-dependent HIF-1 activation

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