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While predominantly expressed in the pancreas, GI tract, and proximal digestive tract where it exerts many of its effects on metabolism, GLP1R has also been detected in normal human thyroid, brain, breast, muscle, and skin tissue (18, 19)

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For instance, AKT kinase phosphorylates the Akt substrate of 160 kDa (AS160) which triggers the translocation of the glucose transporter GLUT4 to the cell surface, thus enhancing glucose uptake [50] (Fig

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