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In this review, we describe recent discoveries providing evidence for the contribution of the GHGHRIGF1 axis on mitochondrial biogenesis, mitophagy (or autophagy), and mitochondrial function under multiple physiological conditions

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Earlier functional studies using inhibition of cAMP formation as an endpoint showed that monensin (a drug that inhibits endosomal recycling), truncated MOR mutants (Qiu et al., 2003) or MOR splice variants (Koch et al., 2001