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Buthionine sulfoximine (BSO) treatment leads to depletion of intracellular glutathione levels and induces autophagy in spermatogonia and retinal pigment epithelial cells [12, 13]

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BPC-157 and TB-500 are commonly studied in controlled laboratory settings involving peptide signalling, cellular migration models, extracellular matrix systems, angiogenesis-related research, cytoskeletal pathway models, and tissue-remodelling investigations