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Umkehrer, C

Ahn (Johns Hopkins University) for study administration and data collection

Notably, potential biomarkers such as 11,12-epoxyeicosatrienoic acid (AUC = 0.964) and MG (PGF2alpha/0:0/0:0) (AUC = 0.953) are implicated in pathophysiological mechanisms of MMA and its standard treatment through their association with inflammation, oxidative stress, and altered fatty acid metabolism

Microglial TLR9 stimulation increases production of pro-inflammatory cytokines and chemokines, while astrocytic activation contributes to gliosis and release of neurotoxic mediators

These do not represent typical outcomes, and results vary

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