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In experimental models of T2DM, EPO administration ameliorates cognitive deficits by mitigating cerebral iron overload, suppressing the ferroptosis pathway, and modulating the expression of key regulatory proteins such as GPX4 and ferritin

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Acknowledgements We express our gratitude to the Vice-Chancellor for Research, Isfahan University of Medical Sciences for financial support through the scientific code 398979 and grant number 1989096

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