In states of insulin resistance, hepatic steatosis, and renal impairment, mitochondrial electron transport chain dysfunction drives the excessive production of ROS, which results in redox imbalance and metabolic stress (96, 97)
The peaks of 2-S-pyridinium derivatives of N-Hcy-Lys, Cys, Hcy, and GSH were monitored at 320 nm
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On the second day after FA injection, samples of kidney specimens and blood were harvested for further examination