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Patient-centred approaches for the management of unpleasant symptoms in kidney disease

While mechanistic studies suggest that VEs antioxidant properties attenuate lipid peroxidation cascades and mitochondrial dysfunction, emerging evidence positions VE as a pleiotropic regulator, suppressing glutamate excitotoxicity, stabilizing BBB integrity, and tempering IL-1-mediated neuroinflammation (Fig

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GM-CSF during remission induction for acute myelogenous leukemia

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