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Neuroproteomic study of nitrated proteins in moderate traumatic brain injured rats treated with glutamyl cysteine ethyl ester administration post injury: insight into the role of glutathione elevation in nitrosative stress

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Elevated intracellular Ca 2+ activates Ca 2+ -sensitive Gardos channels (KCa3.1, encoded by KCNN4), resulting in K + efflux, membrane hyperpolarization, Cl loss, and subsequent osmotic cell shrinkage (Qadri et al., 2012

However, more studies are needed to determine the optimal dosage, administration method, and long-term effects of glutathione therapy

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