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To clear inflammatory sources such as exogenous inflammatory sources (e.g., bacterial viruses) and endogenous pro-inflammatory sources (e.g., damaged organelles, aggregates), autophagic cargoes are usually regulated by ubiquitin and are regulated by a type called SLR (sequestrate-like receptor: p62 [SQSTM1], NBR1, OPTN, NDP52, TAX1BP1, etc.) (143)