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During the aging process, L-carnitine administration caused an increase in VGLUT-1, VGLUT-2, EAAT-1, EAAT-2, EAAT-3 levels in hippocampus tissues

How can we reconcile differences in preclinical toxicity and contrasting conclusions arising from the studies of Lankas et al (155) and Burkey et al (156) regarding the potential importance of DPP8/9 as mediators of off-target (non-DPP4-mediated) toxicity with specific enzyme inhibitors

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This combination of pathway activity is under investigation in preclinical laboratory models for its ability to modulate downstream signaling cascades and molecular interactions

At the same time, rare side effects continue to be identified