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G6PDH-generated NADPH regulates oxidative protein folding Given that GSEA suggests a progressive, differentiation-related malfunctioning in G6pdh chon growth plates, we first focused on understanding the link between NADPH-mediated GSSG recycling and protein processing in ER, as this pathway was most increased in the resting chondrocytes (Extended Data Fig

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Blood and urine sample collection The study participants underwent forearm venipuncture overnight, and blood was drawn into ethylenediaminetetraacetic acid (EDTA) anticoagulation tubes

Finally, the strain MG12345 was constructed by deleting glgB (Table 1)