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Taking arginine metabolism as an example, myeloid cells in the tumor microenvironment, including tumor-associated macrophages and myeloid-derived suppressor cells, often suppress effector immunity through ARG1/ARG2-mediated arginine depletion

Although the above discussed studies suggested a considerable association between the selenium-induced GPx level and the radiation resistance of cultured cells and murine models, it can be argued that the direct cause and effect relationship between GPx activity/expression and radiation toxicity was lacking in these studies