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What all of these have in common is excess melanin, the pigment your skin produces in response to various triggers
Redox homeostasis genes modulate response to APR-246 in a p53-dependent and p53-independent manner Previous studies have characterized APR-246 as a modulator of redox homeostasis and inducer of ROS through depletion of key antioxidants, resulting in both p53-dependent and -independent cell death pathways such as ferroptosis [26,27,28,29]
In diseases like cardiomyopathy, and psoriasis it has been shown that increased CPT1 expression is directly correlated with disease state 14, 15
doi: 10.1007/s00125-017-4315-2 135 HeiseTMariADeVriesJHUrvaSLiJPrattEJet al
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