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doi: 10.1038/s41598-017-09898-4

supporting the feasibility of treat-smart personalized therapy rather than non-specific escalation

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In the 1990s, exendin-4, a GLP-1 receptor agonist (GLP-1 RA) structurally similar to endogenous GLP-1, was isolated from Gila monster venom, which subsequently led to the approval of exenatide for type 2 diabetes mellitus (T2DM) management in 2005 [2]

this allows translocation of dietary antigens, intact bacteria and bacteria toxins into systemic circulation, as well as microbiota metabolites (such as LPS and peptidoglycan) in smaller molecules (Han et al., 2025