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Under oxidative stress, certain cysteine residues on KEAP1 undergo chemical modifications that decrease NRF2 binding affinity, thus facilitating the expression of protective redox-related genes [89]

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Acknowledgments This research was supported by a Grant-in-Aid for Exploratory Research (grant number 16K20081) from the Ministry of Education, Culture, Sports, Science, and Technology of Japan, Tokyo, Japan

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