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No, individuals with Wilson disease or other copper metabolism disorders should avoid GHK-Cu entirely

Within pathological settings, the critical link between metabolic reprogramming and immunocyte function becomes most apparent

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Endocrinology & Metabolism, 140(12), 5616-5621

Thus, observed potent induction of ferroptosis, GPX4-dependent novel suppression of mTOR pathway and DNA damage repair response in preclinical in vitro model of TC supports GPX4 targeting for therapeutic benefit in advanced therapy-resistant thyroid cancers