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Herpes simplex virus type 1 infection induces oxidative stress and the release of bioactive lipid peroxidation by-products in mouse P19N neural cell cultures

The concern is physiologically plausible

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This latter, which interacts with the histone acetylase p300 favouring epigenetic positive control of gene transcription, has been shown to act as a transcriptional co-activator of Nrf2 (Figure 3) [26]

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