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In contrast, GLP1/GIP therapy was unable to consistently reduce body weight, food intake, or fat mass in PWA mice over the study period (Fig

Induction of this biosynthetic pathway enhances intracellular GSH levels and promotes cancer cell resistance to ferroptosis, 142,143 whereas pharmacological inhibition of GSH synthesis by L-buthionine-(S,R)-sulfoximine (BSO), a GCL inhibitor, induces ferroptosis 144

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